This site is intended for healthcare professionals

Sign in
Go to /sign-in page

You can view 5 more pages before signing in

Clinical features

Last reviewed dd mmm yyyy. Last edited dd mmm yyyy

Authoring team

Possible features:

  • sudden and profound, painless loss of vision; in branch occlusion, visual defect corresponds to branch affected
  • affected pupil is poorly reactive to light, if at all; consensual light reaction is normal
  • retina is pale and opaque due to oedema
  • retinal arteries are irregular and appear as red threads
  • lacking the inner retinal layers, the fovea remains transparent and presents a conspicuous "cherry red spot" due to transmission of the choroidal circulation
  • optic atrophy develops after a few weeks, the optic nerve becoming dead white

Occlusion due to vasospastic disease is transient, and vision usually returns within a few hours. Occlusion by emboli may be preceded by episodic flickering as minute emboli interrupt blood flow before disintegrating and passing to the periphery.

Related pages

Create an account to add page annotations

Create a free account

Annotations allow you to add information to this page that would be handy to have on hand during a consultation. E.g. a website or number. This information will always show when you visit this page.

The content herein is provided for informational purposes and does not replace the need to apply professional clinical judgement when diagnosing or treating any medical condition. A licensed medical practitioner should be consulted for diagnosis and treatment of any and all medical conditions.

Connect

Copyright 2024 Oxbridge Solutions Limited, a subsidiary of OmniaMed Communications Limited. All rights reserved. Any distribution or duplication of the information contained herein is strictly prohibited. Oxbridge Solutions receives funding from advertising but maintains editorial independence.