This site is intended for healthcare professionals

Go to /sign-in page

You can view 5 more pages before signing in

Aetiology of acute cholecystitis

Last reviewed dd mmm yyyy. Last edited dd mmm yyyy

Authoring team

Biliary obstruction in acute cholangitis is frequently caused by choledocholithiasis. Other causes of acute cholangitis include:

  • benign biliary stricture
  • congenital factors
  • post-operative factors (damaged bile duct, strictured choledojejunostomy, etc.)
  • inflammatory factors (oriental cholangitis, etc.)
  • malignant occlusion - bile duct tumor, gallbladder tumor, ampullary tumor, pancreatic tumor
  • duodenal tumor
  • pancreatitis
  • entry of parasites into the bile ducts
  • external pressure
  • fibrosis of the papilla
  • deuodenal diverticulum
  • blood clot
  • sump syndrome after biliary enteric anastomosis
  • iatrogenic factors
    • acute cholangitis may occur as a result of biliary interventions such as endoscopic retrograde cholangiopancreatography (ERCP) and the use of biliary stents

Note:

  • an increase in incidence is reported in malignant disease, sclerosing cholangitis, and non-surgical instrumentation of the biliary tract as causes of acute cholangitis (1)

Reference:


Create an account to add page annotations

Annotations allow you to add information to this page that would be handy to have on hand during a consultation. E.g. a website or number. This information will always show when you visit this page.

The content herein is provided for informational purposes and does not replace the need to apply professional clinical judgement when diagnosing or treating any medical condition. A licensed medical practitioner should be consulted for diagnosis and treatment of any and all medical conditions.

Connect

Copyright 2024 Oxbridge Solutions Limited, a subsidiary of OmniaMed Communications Limited. All rights reserved. Any distribution or duplication of the information contained herein is strictly prohibited. Oxbridge Solutions receives funding from advertising but maintains editorial independence.